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- Technology Review: Fixing a Genetic Flaw: An international team of researchers has successfully treated dogs with the canine form of Duchenne muscular dystrophy (DMD), a rapidly progressing and ultimately fatal muscle disease that afflicts one out of every 3,600 boys. The researchers used a novel technique called exon skipping to restore partial function to the gene involved in Duchenne. The study, published in Annals of Neurology, gives hope that a similar approach could work in humans.
March 7th
- Differential chromatin marking of introns and expressed exons by H3K36me3 : Abstract : Nature Genetics: exons are preferentially marked with H3K36me3 relative to introns. H3K36me3 exon marking is dependent on transcription and is found at lower levels in alternatively spliced exons, supporting a splicing-relat ed marking mechanism. We further show that the difference in H3K36me3 marking between exons and introns is evolutionarily conserved in human and mouse. We propose that H3K36me3 exon marking in chromatin provides a dynamic link between transcription and splicing.
- Processing the H3K36me3 signature : Abstract : Nature Genetics: A study in Caenorhabditis elegans now shows that nucleosomes covering exons and introns on active genes are differentially marked by H3K36 trimethylation , suggesting a new mode of communication between chromatin and pre-mRNA processing.
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- ExonMiner: Online high-throughpu t Exon array analysis
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- Exon Race Engines » Exon Race Engines
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