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July 4th
- Resveratrol - Linus Pauling Institute at Oregon State University
- Excess Pounds, but Not Too Many, May Lead to Longer Life - NYTimes.com
- More Gene Smart Success Stories | Gene Smart
- The Life Extension Manual - Human Growth Hormone
- SIRT1, neuronal cell survival and the insulin/IGF-1 aging paradox.: Examines reasons for the paradox that while the polypeptide IGF-1 is neuroprotectiv e, defects in IGF-1 signalling extend lifespan.
- FuturePundit: Immune System Rejuvenation May Partially Rejuvenate Brain
- FuturePundit: Wnt Suppresses Stem Cell Repair Work As We Age?: The key to the whole process is Wnt, a protein traditionally thought to help promote maintenance and proliferation of stem cells in many tissues. But in this instance, Wnt appears to block proper communication.
- Teenage 'baby' may lack master ageing gene - health - 25 June 2009 - New Scientist: Pioneering investigations have thrown the first scientific light on the highly unusual case of Brooke Greenberg, a child from Baltimore, Maryland, who is 16 years old, but whose size and development corresponds to that of an infant of 11 months. The hope is that the investigation will throw up new clues to ageing. But the researchers involved say that Brooke's condition is not a reversed version of conditions that lead to premature ageing.
- Stand up and be counted: Deportment may sound dated but it could knock years off your looks | Mail Online: what if someone told you good deportment is the fastest, cheapest beauty treatment you?ll ever come across, that it could knock years off your age, or that mastering the gentle art of better posture would have a profoundly positive effect on all aspects of your life, physical and mental and emotional? Not so old-fashioned sounding now, is it?
- Blood stem cell growth factor reverses memory decline in mice « Biosingularity: A human growth factor that stimulates blood stem cells to proliferate in the bone marrow reverses memory impairment in mice genetically altered to develop Alzheimer?s disease, researchers at the University of South Florida and James A. Haley Hospital found. The granulocyte-co lony stimulating factor (GCSF) significantly reduced levels of the brain-clogging protein beta amyloid deposited in excess in the brains of the Alzheimer?s mice, increased the production of new neurons and promoted nerve cell connections.
July 3rd
- How Long Does Wax Last?
- Dr. Oz on Innovations in Regenerative Medicine - Oprah.com
- io9 - Commonly-Presc ribed Drug Significantly Reduces Memory Loss from Alzheimers - Cure for altzheimers: A growth hormone frequently prescribed for cancer patients has been found to significantly reduce memory loss from Alzheimers disease. The drug, which stimulates the production of red blood cells, essentially washes plaques out of the brain. Plaques are protein buildups (pictured) found in the brains of people suffering from age-related dementia, and are believed to be related to memory loss and other symptoms of Alzheimers. These can be signifcantly reduced if patients take drugs containing granulocyte-co lony stimulating factor (GCSF), a human hormone that stimulates stem cells in bone marrow to produce more red blood cells. Scientists at University of South Florida and James A. Haley Hospital discovered in tests on mice that the surge in new blood cells acts as a purging agent, washing through the brain and cleansing neurons "clogged& quot; by protein buildup. There was ultimately a 32-40 percent reduction in this dementia-causi ng protein, known as beta amyloid, in the mouse brains.
- Inflammaging as a major characteristic of old peop...[Nutr Rev. 2007] - PubMed Result: Widespread aging at the population level is a recent phenomenon that emerged in affluent societies. Inflammation is necessary to cope with damaging agents and is crucial for survival, particularly to cope with acute inflammation during our reproductive years. But chronic exposure to a variety of antigens, especially to some viruses such as cytomegaloviru s, for a period much longer than that predicted by evolution, induces a chronic low-grade inflammatory status that contributes to age-associated morbidity and mortality. This condition carries the proposed name "inflamma ging". Centenarians are unique in that, despite high levels of pro-inflammato ry markers, they also exhibit anti-inflammat ory markers that may delay disease onset. The key to successful aging and longevity is to decrease chronic inflammation without compromising an acute response when exposed to pathogens.
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